Bufo Toads

Nan's Nook : Archives : Botanicals : Bufo Alvarius
Posted by: Zoom Jun 25 03, 10:01 PM GMT
I Was wondering if anyone know how to
aquire one of these unique toads? huh.gif

Posted by: Halitosis Jun 25 03, 10:28 PM GMT
I would like some beaucoup fish.

Posted by: nhdevildog Jun 25 03, 10:49 PM GMT
If your looking for the rocky mountain river toads...the kind that make you $#!$. I have them in my reptile catalog. I honestly don't know what a bufo toad is. But then again work with mainly snakes and arachnids

Posted by: dustyclc Jun 26 03, 12:32 AM GMT
Hey the toad you are looking for or at least the one I know about is Bufo alvarius, Colorado River Toad. It is easy to get if you live in Arizona. I have a friend who caughts them all the time. The effect from the taod are interesting and there is a naughsus feeling bleah.gif that goes along with the smoking of the vemon. biggrin.gif I hope this helped a little wink.gif

Posted by: nhdevildog Jun 26 03, 09:26 AM GMT
The venom also can stop your heart. Toad licking as I have heard it called is not safe.

Posted by: nhdevildog Jun 26 03, 09:35 AM GMT
I contacted my supplier. They are out of season right now. I am checking with a couple of people in the business and should be able to get ahold of one for you.

Posted by: lil'MushMan Jun 26 03, 09:44 AM GMT
well ya got to admit. having a psychoactive pet toad would be pretty cool. instead of having a gecko or something you get a toad biggrin.gif . seems to be rather interesting...

though i have never tried licking a toad and i dont think i want to because it is venom is it not? venom is extremely toxic bleah.gif

maybe one day... think.gif

Posted by: dustyclc Jun 26 03, 10:05 AM GMT
I would not lick it and I have heard smoking it is the way to go. THats the way I did it.

Posted by: mjshroomer Jun 26 03, 10:13 AM GMT
You can read about the Bufo alvarius toad with pictures and milking the glands info at my forum in the journals section of this site.

I should mention that many toad sellers ont he internet and in stores really have no idea what kind of toads they peddle. 99% of the toads sold as Bufo alvarius turn out top be Bufo marinus, the most common of the Bufo family.

mjshroomer. and under noc ircumstances, no matter what you may have heard or read.


Posted by: nhdevildog Jun 26 03, 12:43 PM GMT
Totally true MJ.
I am not into the toads as much as the snakes and arachnids. But, I have done some research after reading this post and have a good lead on the real thing. They run anywhere from 125.00 to 150.00 for a 6-8 incher. I am very skeptical about purchasing and selling these animals. I would not be able to knowingly sell one of these animals for someone to ingest its venom. Its not good for the person and its sure as hell not good for the toad. Again I can not tell people how much danger they are in by ingesting the venom from this toad. IT IS VENOM! People have died from it. So you have the basic price for them. I recommend that if one insists on aquiring one....see a reputable dealer. There are a few in Florida, NH, and RI among other places. Good Luck!

Posted by: Samsara Jun 26 03, 12:52 PM GMT
Who had died from it? I have never heard such a thing. Milking the toad is not bad for it either. It will stress it slightly, but definitly does no damage to it. And the venom would be toxic......If you are smart enough to ingest all of it at once. 100 mg of 5 meo DMT would certainly stop your heart. However, only someone extremley stupid would do that. wink.gif

Posted by: nhdevildog Jun 26 03, 02:52 PM GMT
Topic: TOAD TOXINS This is some info I found online. Don't knbow how to post a link so pasted it.
are treated for snake venom poisoning, with 12 to 15 deaths/year. ... Bufo alvarius
This overview assumes that basic life support measures
have been instituted.
A. There are several types of toxic substances found in
toads, including cardioactive agents, catecholamines,
indolealkylamines and non-cardiac sterols. These toxins
are located in the skin and parotid glands and may be
transferred by handling or ingesting a toad's skin.
0.2.3 HEENT
A. Secretions of the toad parotid glands will cause pain
and severe irritation when placed in eyes, nose, and
A. Dogs who have been poisoned with bufagins develop
ventricular fibrillation and symptoms resembling
digitalis poisoning. Vasoconstriction may also be seen.
A. Dyspnea and weakened respirations may be seen.
A. Paralysis and seizures have been reported in both humans
and animals. Many bufagins have local anesthetic
actions, especially on the oral mucosa.
A. Salivation and vomiting were often seen in animals.
These toxins may cause numbness of the oral mucosa if
A. Cyanosis has been seen in poisoned dogs.
A. HALLUCINATIONS: Drug users have been known to smoke the
chopped skins of toads for their hallucinogenic effect.
A. No toxic levels have yet been established for any of the
bufagins. Since many of the other substances are
metabolized rapidly, laboratory analysis is impractical.
A. There are three primary areas of toxicity, the first
involving cardiac glycoside effects, the second, the
pressor effects, and the third, the hallucinogenic
effects. Usually the cardiovascular effects are the
most prominent. Treatment is directed at prevention of
absorption, and monitoring for EKG effects and
hyperkalemia. Lidocaine, a transvenous pacemaker, and
cholestyramine have all been used to treat digitalis-
like poisonings. FAB fragments have not been reported
to be of use in toad poisoning.
B. Hemodialysis has been ineffective in removing cardiac
A. The skin of one toad is sufficient to cause significant
symptoms and even death in both animals and humans.

B. No toxic serum or blood levels have yet been established.
A. There are several types of toxic substances found in the
venom of toads.
1. CARDIOACTIVE SUBSTANCES: Bufagins (bufandienolides) are
cardioactive substances found in toad venom. They have
effects similar to the cardiac glycosides found in
plants. Bufotoxins are the conjugation products of the
specific bufagin with one molecule of suberylargine
(Chen & Kovarikova, 1967). Bufotoxins were originally
isolated from the parotoid glands of toads, but have
since been seen in various plants and mushrooms
(Siperstein et al, 1957; Lincoff & Mitchel, 1977; Kibmer
& Wichtl, 1986).
2. CATECHOLAMINES: There are also several catecholamines
in toad venom. Epinephrine has been found in as high a
concentration as 5% in the venom of several species.
Norepinephrine has also been found (Chen & Kovarikova,
3. INDOLEALKYLAMINES: Chemicals found include several
bufotenines. Bufotenines are organic bases containing
an indole ring and have primarily oxytocic actions and
often pressor actions (Palumbo et al, 1975). Specific
substances include bufothionine, serotonin,
cinobufotenine, bufotenine, and dehydrobufotenine (Chen
& Kovarikova, 1967). Bufotenine is the 5-hydroxy
derivative of N,N,dimethyltryptamine and is a
hallucinogen (Gilman et al, 1985).
4. NONCARDIAC STEROLS: The sterols found in toad venom
include cholesterol, provitamin D, gamma sitosteral, and
ergosterol. They do not appear to have a significant
role in toxicity (Chen & Kovarikova, 1967; Palumbo et
al, 1975).
A. Toads known to contain toxins include:
1. Bufo alvarius
2. Bufo americanus
3. Bufo arenarum
4. Bufo asper
5. Bufo blombergi
6. Bufo bufo
7. Bufo bufo gargarizans
8. Bufo formosus
9. Bufo fowerii
10. Bufo marinus
11. Bufo melanostictus
12. Bufo peltocephalus
13. Bufo quercicus
14. Bufo regularis
15. Bufo valliceps
16. Bufo viridis
A. Toads are found throughout the world, Bufo marinus having
one of the widest distributions.

A. Poisoning by toad toxins is primarily a problem with
animals and may be fatal (Perry & Bracegirdle, 1973).
There have been fatalities in Hawaii, Phillipines, and
Fiji occurring after eating the toads as food (Tyler,
1976; Palumbo et al, 1975). The toxins are located in It must have a very high LD50?
the skin and parotid glands and may be transferred by
handling a toad. A toad that sits in a dog's watering To kill dogs
dish for some time may leave enough toxin to make the pet
ill (Smith, 1982). The toxicity varies considerably by
the toad species and its geographic location. The death
rate for untreated animals exposed to Bufo marinus is
nearly 100% in Florida, is low in Texas, and only about
5% in Hawaii (Palumbo et al, 1975).
2.3.2 EYES
A. IRRITATION: If the secretions of the toad parotid
glands come in contact with human eyes, pain and severe
irritation will result (Tyler, 1976; Smith, 1982).
2.3.4 NOSE
A. IRRITATION: Exposure of the nasal mucous membranes to
the toad toxins may produce severe irritation (Chen &
Kovarikova, 1967).
2.3.5 THROAT
A. The mouth and throat may become anesthetized if
bufotoxins have been ingested (Chen & Kovarikova, 1967).
A. VENTRICULAR FIBRILLATION: Dogs intentionally poisoned
with bufagins orally develop ventricular fibrillation and
if untreated - death (Palumbo et al, 1975). The symptoms
resemble digitalis poisoning.
B. VASOCONSTRICTION: Bufagins constrict arterial blood
vessels (Chen & Kovarikova, 1967). Bufotenine itself is
not hallucinogenic, but acts as a pressor rather than a
hallucinogen in humans (Kantoretal, 1980).
A. DYSPNEA: Weakened respirations may be seen if toad
toxins have been ingested (Smith, 1982).
A. PARALYSIS: Paraplegia has been noted in toad poisonings
of dogs and cats. Incoordination and progressive
paralysis may be earlier symptoms (Perry & Bracegirdle,
1973; Smith, 1982).
B. SEIZURES: Have been reported in poisoned dogs and a few
cats (Palumbo et al, 1975; Chen & Kovarikova, 1967), as
well as a 5-year-old boy (Hitt & Ettinger, 1986). Onset
was within 5 minutes. The seizures continued unabated
for 60 minutes.
C. LOCAL ANESTHESIA: Many bufagins have local anesthetic
actions, especially on the oral mucosa (Chen &
Kovarikova, 1967).
A. SALIVATION: Intense salivation is usually seen in
poisoned cats and dogs (Perry & Bracegirdle, 1973), and

was seen in one 5-year-old boy (Hitt & Ettinger, 1986).
B. VOMITING: Is often present in animals (Perry &
Bracegirdle, 1973).
C. NUMBNESS: If ingested, the toxins cause numbness of the
oral mucosa (Smith, 1982; Chen & Kovarikova, 1967).
A. HYPERKALEMIA: Similar to that seen with digitalis
poisoning, may be seen.
A. FEVER: Is a symptom common to ingestion of toads by cats
and dogs (Perry & Bracegirdle, 1973).
A. CYANOSIS: Has been seen in dogs (Hitt & Ettinger, 1986).
A. PERSPIRATION: Although handling toads is generally not
considered seriously injurious to humans, it is thought
to dramatically reduce perspiration (Smith, 1982).
A. HALLUCINATIONS: In 1971, drug users in Queensland were
smoking the chopped skins of Bufo marinus for its
hallucinogenic effect (Tyler 1976). Toad skin has been
used for its hallucinogenic properties throughout the
world (Emboden, 1979), but Bufo alvarins is the only Bufo
species known to contain a hallucinogenic tryptamine
(McKenna & Towers, 1984).
A. No toxic levels have yet been established for any of the
bufagins. Many of the other substances are metabolized
rapidly, and laboratory analysis would be impractical.
3.2.3 OTHER
A. EKG: Patients who have had significant exposures should
have a baseline EKG to observe for abnormalities.
Symptomatic patients should continue to have EKGs
B. A serum potassium level should be drawn to test for
hyperkalemia (Chen & Kovarikova, 1967).
A. A typical animal case report involves a dog that finds a
slow hopping toad and mouths the animal playfully. The
animal usually experiences immediate salivation, and
irritation of the mucus membranes of the mouth and
throat. If the dog eats the toad, vomiting and paralysis
may lead to seizures and death. Animals who recover
usually do not have significant sequelae.
B. Although human deaths have been reported in the lay
literature, we were able to find only one case report of
a human death or serious intoxication in the medical
literature. This was a 5-year-old who had mouthed a Bufo
alvarius (Colorado River Toad) and developed status
epilepticus successfully treated with diazepam and
phenobarbital (Hitt & Ettinger, 1986).

Support respiratory and cardiovascular function.
A. There are 3 primary areas of toxicity. The first
involves the cardiac glycoside-like effects of the
bufagins; the second is the pressor effects of the
catecholamines; and the third is the hallucinogenic
effect of the indolealkylamines. After a toad had been
ingested, it is difficult to evaluate which of these
effects will predominate. Usually, the cardiovascular
effects are the most prominent. The patient should be
observed for arrhythmias and for hallucinations. There
have been minimal human exposures, so clinical
presentation and course are difficult to predict.
1. Emesis may be indicated in substantial recent
ingestions unless the patient is obtunded, comatose or
convulsing or is at risk of doing so based on
ingestant. Emesis is most effective if initiated
within 30 minutes of ingestion. Dose of ipecac syrup:
kilograms): 30 milliliters; CHILD 1 TO 12 YEARS: 15
milliliters; CHILD 6 TO 12 MONTHS (consider
administration in a health care facility): 5 to 10
milliliters. After the dose is given, encourage clear
fluids, 6 to 8 ounces in adults and 4 to 6 ounces in a
child. The dose may be repeated once if emesis does
not occur within 30 minutes.
2. If emesis is unsuccessful following 2 doses of ipecac,
the decision to lavage or otherwise attempt to
decontaminate the gut should be made on an individual
basis. This amount of ipecac poses little toxicity of
3. Refer to the IPECAC/TREATMENT management for further
information on administration and adverse reactions.
1. Cardiac glycosides and bufandienolides are adsorbed to
activated charcoal and enterohepatic circulation may be
decreased by multiple-dose activated charcoal (Balz &
Bader, 1974).
2. Repeated oral charcoal dose (every 2 to 6 hours) may
enhance total body clearance and elimination. A saline
cathartic or sorbitol may be given with the first
charcoal dose and repeated until charcoal appears in
the stools. Do not repeat charcoal if bowel sounds
3. Administer charcoal as slurry. The FDA suggests a
minimum of 240 milliliters of diluent per 30 grams
charcoal (Dose: Optimum dose of charcoal is not
established; usual INITIAL dose is 30 to 100 grams in
adults and 15 to 30 grams in children; some suggest
using 1 to 2 grams per kilogram as a rough guideline,
particularly in infants). REPEAT doses have ranged
from 20 to 50 grams in adults. Doses in children have

not been established, but one-half the initial dose is
4. Administer a saline cathartic or sorbitol, with the
INITIAL charcoal dose, mixed with charcoal or
administered separately. Dose:
a. Magnesium or sodium sulfate (ADULT: 20 to 30 grams
per dose; CHILD: 250 milligrams per kilogram per
dose) OR magnesium citrate (ADULT AND CHILD: 4
milliliters per kilogram per dose up to 300
milliliters per dose).
b. Sorbitol (ADULT: 1 to 2 grams per kilogram per dose
to a maximum of 150 grams per dose; CHILD: (over 1
year of age): 1 to 1.5 grams per kilogram per dose
as a 35 percent solution to a maximum of 50 grams
per dose). Consider administration in a health care
facility, monitoring fluid-electrolyte status,
especially in children.
5. When used with multiple-dose charcoal regimens, the
safety of repeated cathartics has not been established.
Hypermagnesemia has been reported after repeated
administration of magnesium containing cathartics in
overdose patients with normal renal function. In young
children, cathartics should be repeated no more than 1
to 2 times per day. Administration of cathartics
should be stopped when a charcoal stool appears.
Cathartics should be used with extreme caution in
patients who have an ileus or absent bowel sounds.
Saline cathartics should be used with caution in
patients with impaired renal function.
6. Refer to the ACTIVATED CHARCOAL/TREATMENT management
for further information on administration and adverse
C. One of the best first aid measures to prevent toxicity
in animals is to immediately flush the oral mucous
membranes of dogs, cats, and even people who have had
mucous membrane exposure to decrease absorption. Do not
swallow the rinse water.
1. MONITOR EKG CONTINUOUSLY: For abnormal cardiac rates
and rhythms. In patients with previously healthy
hearts, the most common manifestation is bradycardia
with or without varying degrees of AV block. Peaked T
waves, depressed ST segments, widened QRS, and
prolonged PR interval may also be noted.
2. HYPERKALEMIA: Hyperkalemia following acute overdose
may be life-threatening. The emergency management of
life-threatening hyperkalemia (potassium levels greater
than 6.5 mEq/L) includes the intravenous administration
of bicarbonate, glucose, and insulin. DOSE:
Administer 0.2 units/kg of regular insulin with 200 to
400 mg/kg glucose (IV dextrose 25% in water).
Concurrent administration of IV sodium bicarbonate
(approximately 1.0 mEq/kg up to 44 mEq per dose in an
adult) may be of additive value in rapidly lowering

serum potassium levels. Monitor the EKG while
administering the glucose, insulin, and sodium
bicarbonate. This therapy should lower the serum
potassium level for up to 12 hours.
3. ATROPINE: Atropine is useful in the management of
bradycardia, varying degrees of heart block and other
cardiac irregularities due to the digitalis-like
induced effects of enhanced vagal tone on the SA node
rhythmicity and on conduction through the AV node.
DOSE: Adult: 0.6 mg per dose IV; Child: 10 to 30
mcg/kg per dose up to 0.4 mg per dose (may be repeated
as needed to achieve desired effects). Monitor EKG
carefully while administering atropine.
4. PHENYTOIN: Phenytoin is useful in the management of
digitalis-like induced ventricular dysrhythmias and
improves conduction through the AV node. Low dose
phenytoin (Adult: 25 mg per dose IV at 1 to 2 hour
intervals; Child: 0.5 to 1.0 mg/kg per dose IV at 1 to
2 hour intervals) appears to improve AV conduction.
Larger doses are needed for the management of
ventricular dysrhythmias: Loading Dose for adults and
children: Administer 15 mg/kg up to 1.0 gram IV not to
exceed a rate of 0.5 mg/kg per minute. Maintenance
Dose: Adults - administer 2 mg/kg IV every 12 hours as
needed; Child - administer 2 mg/kg every 8 hours as
needed. Monitor serum phenytoin levels just prior to
initiating and during maintenance therapy to assure
therapeutic levels of 10 to 20 mcg/ml (39.64 to 79.28
nmol/L). Monitor EKG carefully.
a. Lidocaine is useful in the management of ventricular
tachyarrhythmias, PVC's, and bigeminy. Lidocaine does
not improve conduction through the AV node.
b. ADULT: BOLUS: 50 to 100 milligrams (0.70 to 1.4
milligrams per kilogram) under EKG monitoring. Rate:
25 to 50 milligrams per minute (0.35 to 0.70
milligrams per kilogram per minute). A second bolus
may be injected in 5 minutes if desired response is
not obtained. No more than 200 to 300 milligrams
should be administered during a one hour period.
INFUSION: Following a bolus, an infusion at 1 to 4
milligrams per minute (0.014 to 0.057 milligram per
kilogram per minute) may be used. PEDIATRIC: BOLUS:
1 milligram per kilogram. INFUSION: 3 micrograms per
kilogram per minute.
6. TRANSVENOUS PACEMAKER: Insertion of a transvenous
pacemaker should be considered in those patients with
severe bradycardia and/or slow ventricular rate due to
second degree AV block who fail to respond to atropine
and/or phenytoin drug therapy.
7. FAB FRAGMENTS: Have not been documented to be of any
value in the treatment of bufagins. Cross reactivity
has not been proven.
8. CHOLESTYRAMINE: Digitoxin (and theoretically bufagins)
elimination appears to be enhanced by the serial

administration of cholestyramine, 4 grams orally every
6 hours. Cholestyramine appears to have minimal effect
on absorption and excretion of cardiac glycosides in
9. One 5-year-old boy did well on high-dose hydrocortisone
sodium succinate and phenobarbital (Hitt & Ettinger,
B. ANIMALS (ESPECIALLY DOGS) (Palumbo et al, 1975):
1. ATROPINE: May be used to decrease secretions and block
vagal effects. It is not a specific antidote.
effects of bufotoxins on the mucous membranes of the
mouth and other organs, but have little direct action.
tolerance to toad venom intoxication.
4. PROPRANOLOL: Has been tried on canines, with some
success. The dose used was high: 5 mg/kg.
It has been used after IV administration of methyl
proscillaridin (Belz & Bader, 1974).
B. HEMODIALYSIS: Has been ineffective in removing cardiac
glycosides but may assist in restoring potassium to
normal levels. It has yet to be tried on bufagins.
A. Wash exposed area extremely thoroughly with soap and
water. A physician may need to examine the area if
irritation or pain persists after washing.
A. Effects may be seen after dermal exposure. Treatment
should be as appropriate under the oral treatment
A. The skin of one toad is sufficient to cause significant
symptoms and even death in both animals and humans.
A. No toxic serum or blood levels have yet been established.
6.6 LD50/LC50
NAME Mean (Geo.)
LD,, mg/kg
Arenobufagin 0.08
Bufotalin 0.13
Desacetylbufotalin 0.26
Cinobufagin 0.20
Acetylcinobufagin 0.59
Desacetylcinobufagin inactive
Cinobufotalin 0.20
Acetylcinobufotalin 0.18
Desactylcinobufotalin inactive
Marinobufagin 1.49
Acetylmarinobufagin 0.95
12Beta-Hydroxymarinobufagin 3.00

Bufotalidin (hellebrigenin) 0.08
Acetylbufotalidin 0.06
Resibufogenin inactive
Acetylresibufogenin inactive
12Beta-Hydroxyresibufogenin 4.16
Bufalin 0.14
Telocinobufagin 0.10
Bufotalinin 0.62
Artebufogenin inactive
Gamabufotalin 0.10
Vallicepobufagin 0.20
Quercicobufagin 0.10
Viridobufagin 0.11
Regularobufagin 0.15
Fowlerobufagin 0.22
NAME Mean (Geo.)
LD, mg/kg
Viridobufotoxin 0.27
Vulgarobufotoxin 0.29
Cinobufotoxin 0.36
Gamabufotoxin 0.37
Arenobufotoxin 0.41
Marinobufotoxin 0.42
Regularobufotoxin 0.48
Alvarobufotoxin 0.76
Fowlerobufotoxin 0.79
C. REFERENCE: (Chen & Kovarikova, 1967).
A. The structure of the cardioactive bufadienolides leads to
greater potency than the corresponding plant glycosides
thus the cardenolides of plants - digitoxigenin,
periplogenin, oleandrigenin, sarmentogenin, and
strophanthidin, corresponding to bufalin,
telocinobufagin, bufotalin, gamabufotalin, and
bufotalidin - have lower toxicities.
B. The toxicity of the cardioactive bufotoxins is lower than
those of the corresponding bufagins (bufadienolides)
(Chen & Kovarikova, 1967).
C. The skin of Bufo alvarius contains 5-methoxy-N,N-
dimethyltryptamine (5-MeO-DMT) at a concentration of 50
to 160 mg/g of skin (Daly & Witkop, 1971).
A. BUFOTOXINS: Is the name of a collection of compounds
found in the toad venom which may be secreted into toad
skin or found in 2 glands behind the eyes, called parotid
glands (Tyler, 1976). Bufotoxins may also be
specificially applied to the conjugates of a bufagin with
B. Before digitalis was extracted from Digitalis purpura,
dried and powdered toad skins were used as a cardiac
medication (Burton, 1977). Other "folk" uses include
expectorant, diuretic, and remedy for toothaches,
sinusitis, and hemorrhage of the gums.
C. Toad skins have also been used for their hallucinogenic

effect (Emboden, 1979).
A. The oral absorption of the bufagins and bufotoxins is
generally poor. Less than 15% of cinobufagin is absorbed
orally in rats.
B. Other components of toad venom are rapidly absorbed via
mucous membranes and cause immediate symptoms in animals
(Smith, 1982).
8.4.3 BILE
A. Little could be found concerning the excretion of these
compounds; similar cardenolides and substances such as
proscillaridin are excreted largely in the bile (Belz &
Bader, 1974).
A. Most bufandienolides are cardiotonic sterols synthesized
by toads from cholesterol (Siperstein, 1957). The
lactone ring is 6-membered of an alpha pyrone type
attached to C17. They have a secondary hydroxy group at
C3 and are called bufagins - which corresponds to the
aglycones found in the cardiac glycosides in plants.
None of these bufandienolides conjugates with a
carbohydrate (as do the plants) to form glycosides, but
some do form bufotoxins by combining with suberylargine
(Chen & Kovarikova, 1967).
B. In the toad, some of these compounds (eg, resibufogenin)
are ouabain-like and increase the force of contraction of
heart muscle (Lichtstein et al, 1986).
C. The pharmacology of the catecholamines found in toad
venom is well known and need not be discussed here.
D. INDOLEALKYLAMINES: Pharmacology is also known. Besides
having some hallucinogenic effects, these compounds may
stimulate uterine and intestinal muscle (Chen &
Kovarikova, 1961).
A. Bufagins and bufotoxins have been shown to inhibit
sodium, potassium, ATPase activity (Lichtstein et al,
1986). Their action is almost the same as that of the
digitalis glycosides (Palumbo et al, 1975).
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levels of intravenous methyl proscillaridin. Klin
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tryptamine (a, O-dimethylserotonin), a hallucinogenic
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A. Written by: David G. Spoerke, M.S., RPh., 06/86
B. Reviewed by: Ken Kulig, M.D., 06/86
C. Specialty Board: Biologicals
D. In addition to standard revisions of this management
certain portions were updated with recent literature:

Posted by: Toad Jun 26 03, 04:23 PM GMT
This Toad doesn't mind being licked, and I'm UNIQUE. biggrin.gif

Posted by: Bobkat Jun 26 03, 05:28 PM GMT
Hey Toad, do you have the album "JACK THE TOAD" by Savoy Brown?

Posted by: Zoom Jun 26 03, 06:25 PM GMT
From what I understand no one acually "licks"
the toad. laugh.gif

The milk is dried and smoked in a VERY
small amount. About the size of a match
head. Contains 15% 5-meo-dmt. wink.gif

Anyone live in the Arizona/Colorado
river area? biggrin.gif

Posted by: nhdevildog Jun 26 03, 06:44 PM GMT
Licking is just a figure of speech. I think I saw it on the Simpsons or something one time too.

Posted by: CharlieBrown Jun 26 03, 06:45 PM GMT

If you're just looking for the buzz, try that link. The toad is something that you'll have to feed, take care of, and respect for the potentially dangerous animal it is. I'm not saying one shouldn't own them, but there definitely needs to be serious thought put to it before making the decision to buy one.
Just my $0.02


Edit* They really are a cool animal, and I plan to own one some day. I'm just turning people on to the pure 5-MeO if they don't know already. smile.gif

Posted by: Rasputin Jun 26 03, 06:46 PM GMT
Don't miss Bufo Alvarius: Sonoran Desert Toad guys. MJ always has some pretty solid info.

Posted by: groingrinder Jun 27 03, 03:00 AM GMT
Wow Bobkat I haven't heard anyone mention Savoy Brown since the seventies. I always wanted to see a concert with Savoy Brown and Brownsville Station.

YOU DO NOT LICK TOADS. Toad venom is smoked in glass pipes. In just a few more months I will be going on a TOAD ROUNDUP, after getting my fishing license with valid reptile stamp, of course. These little suckers hibernate in the dried mud until the monsoon rains start coursing through the washes, then they magically wake up and wander around going "MYAAAAAAAAAAAAAAAAA, MYAAAAAAAAAAAAAAAAA". Don't fear they will come to no harm, as they will be milked and turned loose. A friend suggested using a syringe to suck out the venom from the glands instead of milking them, but I believe that will hurt the toads and have questionable success.

Posted by: mjshroomer Jun 27 03, 04:49 AM GMT
The only toad that gets you high is the Bufoi alvarius from Arizona normally knonw as the Sonoran River Toad.

It 's range is from Arizona to Mexico.

Please do not lick or smoke any of the other Bufo toads, espeially the common toad Bufo marinus. This toad is common in Hawaii where i Lived for 12 years. I have seen dogs poisoned by this by just putting a toad in their mouth. It is not a pretty sight to watch.


Many unscrupulous dealers in Amsterdam and Toad sellers in most pet stores which sell exotic reptiles and toads have no idea what toads they really have. I have met several people over the yrears hwo have Bufo marinus toads sold to them as toads with five meo-DMT intheir glands.

And again. No mattrer how many articles asay colelge kids caught licking toads on campus. Do not believe them. Unless you go tot he Sonoran Desert

Posted by: jezu Jun 27 03, 09:11 PM GMT
Ok then so has any one here actualy done kermit ? laugh.gif

Posted by: Nanook Jun 27 03, 11:19 PM GMT
Excellent thread.

I kept an association with a amateur herpatologist a number of years and have frequently kept snakes and reptiles as pets.

My friend had a federal importers license and imported many venomous reptiles. He got in a box of cobras at the airport from Thailand.

I sat in his basement as he milked the snakes. I swear this guy was half... No 3/4 insane. No gloves, he would open a cage with a dozen hooded, hissing, really pissed cobras... They waved back and forth like grass in a breeze, a hypnotizing effect... He would stand an watch, pick his time, and dart his hand in and bring out a snake holding it with the head pinched between his fingers... After drinking 4-5 beers ohmy.gif

Then he would milk the snakes, one at a time, into a shot glass covered with a condom and collect the venom in a syringe.

Dried venom could be smoked, it would wack your head. Wacked mine, like smoking a general anesthetic, all numb and tingly wacko.gif

So another friend of mine, 7/8ths crazy, buys a couple of snakes with the idea he has himself a bedroom high for the cost of a couple mice a week. But two snakes are not enough to keep a teenaged head like that buzzing, so he looked for a more potent delivery method.

He dried the cobra venom and snorted a line. As soon as the venom contacted the mucous membranes he went into immediate respiratory arrest. His diaphram siezed.

My friend was alone in the house, he said the venom burned so bad in his sinuses that it was like flames, and he could not take a breath. He knew he was dying, he went to the bathroom, turned on faucet in the tub, and cupping the water out of the faucet with his hands, used the water pressure to flood his sinuses, dilute and rinse out the venom. He said water entering his lungs unsiezed his diaphram and he was able to exhale.

He narrowly missed a trip to the mortuary. 3-5 minutes in a condition like that is all the longer you can remain consious.

Just don't get stupid with reptile venom ok? wink.gif

Posted by: dustyclc Jun 28 03, 12:32 AM GMT
Amen to that Nan. I have smoked the vemon of Bufo alvarius and it is interesting to say the least. I would not tell everyone to smoke it but it is a unique experience. biggrin.gif

Posted by: Nanook Jun 28 03, 01:25 AM GMT
I don't see any problem with smoking it, a lot of people do. And toads are a lot safer than cobras. It's talk of licking and people exploring alternate delivery methods (like snorting) that have me saying there is a real and true danger in reptile venom.

Posted by: dustyclc Jun 28 03, 02:38 AM GMT
I see and hear what you are saying and I only have to add that if one does there research and learns from the old teaches the methods they used one will usually be OK. Read Read Read and Read some more.

Posted by: Zoom Jun 28 03, 11:28 AM GMT
I hear ya Dusty wink.gif

Knowledge is power.
Didn't know people smoked
snake venom. ohmy.gif

Dont think I'd go that route. bleah.gif

Does anyone know how someone on the
east coast could aquire one? Are they
protected or illegal? huh.gif

I can't find one for sale anywhere. mad.gif

Posted by: groingrinder Jun 28 03, 12:56 PM GMT
..........and don't inject it into your eyeball with a syringe. laugh.gif laugh.gif laugh.gif

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